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  1. We propose two new haplotype-sharing methods for identifying disease loci: the haplotype sharing statistic (HSS), which compares length of shared haplotypes between cases and controls, and the CROSS test, whic...

    Authors: Ilja M Nolte, AndrƩ R de Vries, Geert T Spijker, Ritsert C Jansen, Dumitru Brinza, Alexander Zelikovsky and Gerard J te Meerman
    Citation: BMC Proceedings 2007 1(Suppl 1):S129

    This article is part of a Supplement: Volume 1 Supplement 1

  2. Rheumatoid arthritis is a complex disease caused by a combination of genetic, environmental, and hormonal factors, and their additive and/or non-additive effects. We performed a linkage analysis to provide evi...

    Authors: Cheongeun Oh
    Citation: BMC Proceedings 2007 1(Suppl 1):S110

    This article is part of a Supplement: Volume 1 Supplement 1

  3. We proposed a confidence interval method for disease gene localization by testing every position on each chromosome of interest for its possibility of being a disease locus and including those not rejected int...

    Authors: Shuyan Wan and Shili Lin
    Citation: BMC Proceedings 2007 1(Suppl 1):S106

    This article is part of a Supplement: Volume 1 Supplement 1

  4. Accounting for interactions with environmental factors in association studies may improve the power to detect genetic effects and may help identifying important environmental effect modifiers. The power of unp...

    Authors: Astrid Dempfle, Rebecca Hein, Lars Beckmann, AndrƩ Scherag, Thuy Trang Nguyen, Helmut SchƤfer and Jenny Chang-Claude
    Citation: BMC Proceedings 2007 1(Suppl 1):S73

    This article is part of a Supplement: Volume 1 Supplement 1

  5. Rheumatoid arthritis (RA) is a complex disease that involves both environmental and genetic factors. Elucidation of the basic etiologic factors involved in RA is essential for preventing and treating this dise...

    Authors: Yi-Shin Chen, Yen-Feng Chiu, Hui-Yi Kao and Fang-Chi Hsu
    Citation: BMC Proceedings 2007 1(Suppl 1):S71

    This article is part of a Supplement: Volume 1 Supplement 1

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