Background
Crohn’s Disease (CD) is a chronic inflammatory bowel disease characterised by recurrent intestinal inflammation [1]. Adipose tissue has metabolic and immune functions regulated through the expression of hormones and cytokines [2, 3]. Conventionally, adiposity in CD is believed to reflect disease activity, nutritional status and possibly corticosteroids. Emerging data suggests that adipose tissue may play a more complex immunoregulatory role in CD [4].